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Tuesday, May 12, 2020 | History

4 edition of Cell death pathways in irradiated prostate cells found in the catalog.

Cell death pathways in irradiated prostate cells

Gillian Bromfield

Cell death pathways in irradiated prostate cells

by Gillian Bromfield

  • 302 Want to read
  • 14 Currently reading

Published by National Library of Canada in Ottawa .
Written in English


Edition Notes

Thesis (M.Sc.) -- University of Toronto, 2002.

SeriesCanadian theses = -- Th`eses canadiennes
The Physical Object
FormatMicroform
Pagination2 microfiches : negative.
ID Numbers
Open LibraryOL19897964M
ISBN 100612740501
OCLC/WorldCa54206062

Phosphorylation of eIF4E by MNKs supports protein synthesis, cell cycle progression and proliferation in prostate cancer cells. Carcinogene – Bjornsti, M.A. and Houghton, P.J. ( a). In deed several evidences have proved that PARP inhibitors sensitize human prostate cancer cell lines [, , ]. It is also know that treatment with high doses of chemotherapy induces massive DNA Author: Luis A. Espinoza.

What causes cancer? I am going to attempt to give the shortest, best, detailed description of cancer ever written. ;-) Ready?! Here goes Cancer is a breakdown in the integrity of the signaling pathways in a . A large body of work strongly supports the existence of crosstalk between cancer and the circadian clock. Indeed, escape from circadian physiologic discipline is relevant in tumorigenesis, where the Cited by: 8.

  Abstract. There are several well-established mechanisms involved in radiation-induced cell death in mammalian cell systems. The pmediated apoptotic pathway is the most widely Cited by: 3. Silibinin, a traditional hepatic protectant, elicits protective effects against UVB-induced cellular damage. In A cells, the insulin-like growth factor-1 receptor (IGF-1R) was markedly up-regulated by UVB .


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Cell death pathways in irradiated prostate cells by Gillian Bromfield Download PDF EPUB FB2

Photodynamic therapy (PDT) is an emerging cancer therapy that uses the combination of non-toxic dyes or photosensitizers (PS) and harmless visible light to produce reactive oxygen species and destroy Cited by: TNF-alpha-mediated apoptosis in normal human prostate epithelial cells and tumor cell lines Article in Cancer Letters (2) February with 32 Reads How we measure 'reads'.

Mitotic Catastrophe. Mitotic catastrophe (MC) is a mechanism of delayed mitotic-linked cell death resulting from aberrant mitosis, although there is an ongoing debate over whether MC is a specific.

However, the schistosome-derived cells/molecules responsible for the strong protective effects in the RA model remain unclear.

The concept of immunogenic cell death (ICD) was proposed to describe. The existence of stem cells (SCs) was firstly demonstrated by James Till and the late Ernest McCulloch in in their earlier work on the radiation sensitivity of mouse bone marrow cells by showing that limited numbers of cells could give rise to clonal colonies of erythroid and myeloid cells in the spleens of the irradiated Cited by: 2.

Prostate cancer co-opts a unique set of cellular pathways in its initiation and progression. The heterogeneity of prostate cancers is evident at earlier stages, and has led to rigorous efforts to Cited by: The development of novel therapies or the improvement of currently used approaches to treat prostate cancer (PCa), the most frequently diagnosed male tumor in developed countries, is an urgent need.

In Author: Federica Rotundo, Denis Cominetti, Rihan El Bezawy, Stefano Percio, Valentina Doldi, Monica Tortoret. Differentiation Therapy. Differentiation is the process by which immature cells develop into the characteristic phenotype of a mature cell. In cancer, differentiation is often characterized by cell cycle Author: William K.

Oh, Mark Hurwitz, Anthony V. D'Amico, Jerome P. Richie, Philip W. Kantoff. All life on earth must cope with constant exposure to DNA-damaging agents such as the Sun's radiation. Highly conserved DNA-repair and cell-cycle checkpoint pathways allow cells to deal.

Normal cells make their own arginine, but 90 percent of sarcoma cancer cells cannot (3). When deprived of arginine, these cells starve, and begin a process called autophagy (self-eating). The highly conserved Cdc6 protein is required for initiation of eukaryotic DNA replication and, in yeast and Xenopus, for the coupling of DNA replication towe show that human.

Ligands that activate the CS-GRP78 signaling pathway can have variable and even opposing effects on cellular proliferation, apoptosis and cell survival as well as metabolism ().Cell surface GRP78, a high Author: Udhayakumar Gopal, Salvatore V. Pizzo. Firat E, Gaedicke S, Tsurumi C, Esser N, Weyerbrock A, Niedermann G.

Delayed cell death associated with mitotic catastrophe in γ-irradiated stem-like glioma cells. Radiat Oncol. ; doi: Author: Marzia Toscano, Silvia Palumbo, Paolo Tini, Clelia Miracco, Giovanni Luca Gravina, Sergio Comincini.

Ayanin diacetate-induced cell death was found to be associated with: (i) loss of inner mitochondrial membrane potential, (ii) the release of cytochrome c, (iii) the activation of multiple caspases, (iv). The cancer stem cell model. The cancer stem cell model, also known as the Hierarchical Model proposes that tumors are hierarchically organized (CSCs lying at the apex (Fig.

3).) Within the cancer. Although CD73 expression had been observed on some tumors, such as melanomas and prostate cancers, and tumor-infiltrating leukocytes (TILs), especially regulatory T cells (Tregs) Author: Miao Yu, Gang Guo, Lei Huang, Libin Deng, Chang-Sheng Chang, Bhagelu R.

Achyut, Madison Canning, Nin. Walsh was Editor-in-Chief from (5th, 6th, 7th, and 8th editions), during which time it expanded to encompass four volumes and 4, pages, becoming the definitive Urology textbook in the world.

In recognition for his efforts, the book. Radiation induces increased cell death in prostate cancer cells treated with RAD A, Annexin V flow cytometry apoptosis assay shows that mTOR inhibition alone does not significantly Cited by:   Targeting Cancer Stem Cells with NonToxic Therapies.

by Jeffrey Dach MD. When Jim was 36 year old, he was diagnosed with acute myeloblastic leukemia with symptoms of fatigue. Title:Garlic and its Active Compounds: A Potential Candidate in The Prevention of Cancer by Modulating Various Cell Signalling Pathways VOLUME: 19 ISSUE: 11 Author(s):Saleh A.

Almatroodi, Mohammed Author: Saleh A. Almatroodi, Mohammed A. Alsahli, Ahmad Almatroudi, Arshad H. Rahmani. The importance of understanding the relationship between the proliferation rate and cell‐death rate is best shown with concrete examples.

For instance, malignant lymphoma is a relatively fast‐growing cancer. However, further investigation of non-apoptotic pathways might provide new therapeutic strategies aimed at inducing the non-apoptotic death of cancer cells.

In the present chapter, apoptotic Cited by: 7.Effects of a novel carbocyclic analog of pyrrolo[2,3-d]pyrimidine nucleoside on pleiotropic induction of cell death in prostate cancer cells with different androgen responsiveness.

Prostate cancer is the most .